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Vitamin C Efficacy Studies



As an indication of how prominent members of the medical trade are prepared to lie and deceive to advance their own agenda, I share this 2014 article by Stephen Barrett, M.D., writing for Quackwatch in the US. https://www.quackwatch.com/01QuackeryRelatedTopics/pauling.html

I cannot say it better than the description by Judge Fromholz in NCAHF v King Bio, which described the authors of Quackwatch best: "Dr. Sampson’s university course presents what is effectively a one-sided, critical view of alternative medicine. Dr. Barrett’s heavy activities in lecturing and writing about alternative medicine similarly are focused on the eradication of the practices about which he opines. It is apparent, therefore, that both men have a direct, personal financial interest in the outcome of this litigation. Based on all of these factors, Dr. Sampson and Dr. Barrett can be described as zealous advocates of the Plaintiff’s position, and therefore not neutral or dispassionate witnesses or experts. In light of these affiliations and their orientation, it can fairly be said that Drs. Barrett and Sampson are themselves the client, and therefore their testimony should be accorded little, if any, credibility on that basis as well."
This pathological liar wrote the featured article knowing full well that the following clinical studies published prior to the article proved him for what he is:

Control of Interstitial Pneumonia by Drip Infusion of Megadose Vitamin C, Dehydroepiandrosterone and Cortisol. http://iv.iiarjournals.org/content/22/2/263.long

Vitamin C kills tumor cells with hard-to-treat mutation. http://www.sciencemag.org/.../vitamin-c-kills-tumor-cells...

The orthomolecular treatment of cancer I. The role of ascorbic acid in host resistance. http://www.sciencedirect.com/.../art.../pii/0009279774900180

Vitamin C in human health and disease is still a mystery? An overview. https://www.ncbi.nlm.nih.gov/pubmed/14498993?dopt=Abstract

The orthomolecular treatment of cancer II. Clinical trial of high-dose ascorbic acid supplements in advanced human cancer. http://www.sciencedirect.com/.../art.../pii/0009279774900192

Supplemental ascorbate in the supportive treatment of cancer: Prolongation of survival times in terminal human cancer. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC431183/

Supplemental ascorbate in the supportive treatment of cancer: Reevaluation of prolongation of survival times in terminal human cancer. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC336151/

Pharmacologic concentrations of ascorbate are achieved by parenteral administration and exhibit antitumoral effects. http://www.sciencedirect.com/.../pii/S0891584909001105

Anti-cancer effect of pharmacologic ascorbate and its interaction with supplementary parenteral glutathione in preclinical cancer models. http://www.sciencedirect.com/.../pii/S0891584911003479

Pharmacologic ascorbic acid concentrations selectively kill cancer cells: Action as a pro-drug to deliver hydrogen peroxide to tissues. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1224653/

Pharmacologic doses of ascorbate act as a prooxidant and decrease growth of aggressive tumor xenografts in mice. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2516281/

Ascorbate exerts anti-proliferative effects through cell cycle inhibition and sensitizes tumor cells towards cytostatic drugs. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3082037/

Pharmacological ascorbate induces cytotoxicity in prostate cancer cells through ATP depletion and induction of autophagy. https://www.ncbi.nlm.nih.gov/pubmed/22205155

Mechanisms of ascorbate-induced cytotoxicity in pancreatic cancer. https://www.ncbi.nlm.nih.gov/pubmed/20068072

Pharmacologic ascorbate synergizes with gemcitabine in preclinical models of pancreatic cancer. https://www.ncbi.nlm.nih.gov/pubmed/21402145

Pharmacologic concentrations of ascorbic acid cause diverse influence on differential expressions of angiogenic chemokine genes in different hepatocellular carcinoma cell lines. https://www.ncbi.nlm.nih.gov/pubmed/19932582

Pharmacologic doses of ascorbic acid repress specificity protein (Sp) transcription factors and Sp-regulated genes in colon cancer cells. https://www.ncbi.nlm.nih.gov/pubmed/21919647

High dose of ascorbic acid induces cell death in mesothelioma cells. https://www.ncbi.nlm.nih.gov/pubmed/20171954

Effect of ascorbic acid and hydrogen peroxide on mouse neuroblastoma cells. https://www.ncbi.nlm.nih.gov/pubmed/22469841

Ascorbic acid: chemistry, biology and the treatment of cancer. https://www.ncbi.nlm.nih.gov/pubmed/22728050

SVCT-2 in breast cancer acts as an indicator for L-ascorbate treatment. https://www.ncbi.nlm.nih.gov/pubmed/22665050

Differential augmentative effects of buthionine sulfoximine and ascorbic acid in As2O3-induced ovarian cancer cell death: oxidative stress-independent and -dependent cytotoxic potentiation. https://www.ncbi.nlm.nih.gov/pubmed/21455570

In vitro screening of synergistic ascorbate-drug combinations for the treatment of malignant mesothelioma. https://www.ncbi.nlm.nih.gov/pubmed/21645609

Pharmacological concentrations of ascorbate radiosensitize glioblastoma multiforme primary cells by increasing oxidative DNA damage and inhibiting G2/M arrest. https://www.ncbi.nlm.nih.gov/pubmed/22342518

Pharmacological ascorbic acid suppresses syngeneic tumor growth and metastases in hormone-refractory prostate cancer. https://www.ncbi.nlm.nih.gov/pubmed/20554995

Enhancement of photodynamic antitumor effect with pro-oxidant ascorbate. https://www.ncbi.nlm.nih.gov/pubmed/22246986https://www.quackwatch.com/01QuackeryRelatedTopics/pauling.html



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